Acute Right Ventricular Failure: Pathophysiology, Aetiology, Assessment, and Management Acute Right Ventricular Failure Pathophysiology: Pressure/volume overload→ RV dilation/dysfunction → Low output & systemic congestion. Aetiology: PE,RV infarction, ARDS, post-cardiac surgery. Assessment: Clinical signs,ECG, biomarkers, POCUS (RV size/function), haemodynamic monitoring. Management: · RV-protective ventilation. · Optimize preload, vasopressors, inotropes. · Mechanical support if refractory. #RV

Acute Right Ventricular Failure: Pathophysiology, Aetiology, Assessment, and Management Acute RV Failure: Key Insights 🫀 Harjola et al., Eur Heart J , DOI: 10.1093/eurheartj/ehaf215 Pathophysiology 🔬: Acute RV failure stems from contractility loss, pressure/volume overload, or ischemia, leading to RV dilation, low cardiac output, and hypoperfusion. RV-LV interplay and neurohormonal surges amplify damage. Aetiology ⚠️: - Common: pulmonary embolism, RV infarction, ARDS, sepsis, post-cardiotomy.

Let’s break down real quick the pathophysiology of Acute Respiratory Distress Syndrome (ARDS):⁣ ⁣ 1️⃣ 𝗜𝗻𝗶𝘁𝗶𝗮𝗹 𝗜𝗻𝗷𝘂𝗿𝘆 𝗮𝗻𝗱 𝗜𝗻𝗳𝗹𝗮𝗺𝗺𝗮𝘁𝗶𝗼𝗻:⁣ ▪️ARDS can be triggered by various direct or indirect insults to the lungs. Direct causes include pneumonia, aspiration, and inhalation injury, while indirect causes encompass sepsis, trauma, and pancreatitis.⁣ ▪️The initial injury leads to the activation of the immune system, causing an influx of inflammatory cells (like neutrophils)